JOURNAL OF PRACTICAL HEPATOLOGY ›› 2016, Vol. 19 ›› Issue (6): 766-769.doi: 10.3969/j.issn.1672-5069.2016.06.039
Zhou Aibin,Zhou Junping.
Received:
2016-05-17
Online:
2016-11-10
Published:
2016-11-28
Zhou Aibin,Zhou Junping.. Cytokines and their signal transduction pathways in pathogenesis of radiation-induced liver injury[J]. JOURNAL OF PRACTICAL HEPATOLOGY, 2016, 19(6): 766-769.
[1] Rahbari NN,Mehrabi A,Mollberg NM,et al. Hepatocellar carcinoma:current management and perspectives for the future. Ann Surg,2011,253(3):453-469. [2] 王丽春,赵连三.肝纤维化的实验动物模型. 中国实验动物学报,2014,12(6):246-249. [3] Bujold A,Dawson LA.Stereotactic radiation therapy and selective internal radiation therapy for hepatocellular carcioma. Cancer Radiother,2011,15(1):54-63. [4] Liang SX,Huang XB,Zhu XD,et al. Dosimetric predictor identification for radiation-induced liver disease after hypofractionated conformal radiotherapy for primary liver carcinoma patients with Child-Pugh Grade A cirrhosis.Radiother Oncol,2011,98(2):265-269. [5] 高东伟,于勇华. 细胞因子与放射性肝损伤. 实用肝脏病杂志,2006,9(4):249-251. [6] Wei C,Lei X,Ainiwaer A,et al. Molecular responses of radiation-induced liver damage in rats.Mol Med Rep,2015,11(4):2592-2600. [7] Chen YX,Zeng ZC,Sun J,et al. Mesenchymal stem cell conditioned medium prevents radiation-induced liver injury by inhibiting inflammation and protecting sinusoidal endothelial cells.Radiat Res,2015,56(4):700-708. [8] Sun X,He Y,Ma TT,et al.Participation of miR-200a in TGF-β1-mediated hepatic stellate cell activation.Mol Cell Biochem,2014,388(1-2):11-23. [9] Yu JL,Li JH,Chengz RG,et al. Effect of matrine on transforming growth factorβ1 and hepatocyte growth factor in rat liver fibrosis model. Asian Pac J Trop Med,2014,7(5):390-393. [10] Kajdaniuk D,Marek B,Borgiel Marek H,et al. Transforming growth factor beta1(TGF-β1)in physiology and pathology. Endokrynol Pol,2013,64(5):384-396. [11] 吴戈,肖蕾,张瑞丽,等. 急性放射性肝损伤大鼠模型的建立与评价. 中国实验动物学报,2015,23(2):178-181. [12] Imaeda M,Ishikawa H,Yoshida Y,et al. Long-term pathological and immunohistochemical features in the liver after intraoperative whole-liver irradiation in rats. J Radiat Res,2014,55(4):665-673. [13] 王云莲,艾尼瓦尔·艾木都拉,肖蕾,等.血清转化生长因子β1在大鼠放射性肝纤维化中的动态表达及其相关性研究.现代生物医学进展,2014,14(30):5829-5832. [14] Bai Q,An J,Wu X,et al. HBV promotes the proliferation of hepatic stellate cells via the PDGF-B/PDGFR-βsignalling pathway in vitro.Int J Mol Med,2012,30(6):1443-1450. [15] 李东,李新宇,龚钰清,等. PDGF-BB、TGF-β1与肝纤维化的相关性研究. 海南医学,2012,23(21):99-100. [16] Velasco-Loyden G,Pérez-Carreón JI,Agüero JF,et al.Prevention of in vitro hepatic stellate cells activation by the adenosine derivative compound IFC 305.Biochem Pharmacol,2010,80(11):1690-1699. [17] Borkham-Kamphorst E,Kovalenko E,Van Roeyen CR,et al.Platelet-derived growth factor isoform expression in carbon tetrachloride-induced chronic liver injury.Lab Inves,2008,88(10):1090-1100. [18] Leask A,Holmes A,Black C M,et al. Connective tissue growth factor gene regulation.Requirements for its induction by transforming growth factor-beta 2 in fibroblasts.J Biol Chem,2003,278(15):13008-130015. [19] 胡云龙,孔丽. TGF-β1,CTGF信号传导通路在小鼠肝纤维化发生中的作用. 基础医学与临床,2012,32(1):66-70. [20] Yu FJ,Dong PH,Fan XF,et al. Down-regulation of angiotensinⅡby shRNA reduces collagen synthesis in hepatic stellate cells.Int J Mol Med,2010,25(5):801-806. [21] Ponmari G,Annamalai A,Gopalakrishnan VK,et al. NF-κB activation and proinflammatory cytokines mediated protective effect of indigofera caerulea Roxb on CCl4 induced liver damage in rats.Int Immunopharmacol,2014,23(2):672-680. [22] 李亚萍,党双锁,翟嵩,等. NF-κB、α-SMA及TGFβ1在实验性肝损伤中的表达. 临床肝胆病杂志,2011,27(11):1207-1210. [23] Kim BH,Yoon JH,Yang JI,et al. Guggulsterone attenuates activation and survival of hepatic stellate cell by inhibiting nuclear factor kappa B activation and inducing apoptosis. J Gastroenterol Hepatol,2013,28(12):1858-1868. [24] 高世乐,胡宗涛,秦峰,等. Smads蛋白家族在大鼠放射性肝纤维化形成过程中的表达.实验动物与比较医学,2015,35(3):188-194. [25] Jazirehi AR,Wenn PB,Damavand M. Therapeutic implications of targeting the PI3kinase/AKT/mTOR signaling module in melanoma therapy.Am J Cancer Res,2012,2(2):178-191. [26] Pal I,Mandal M. PI3K and AKt as molecular targets for cancer therapy:current clinincal outcomes. Acta Pharmacol Sin,2012,33(12):1441-1458. [27] 罗晓旭,肖蕾,吴戈,等. PI3K/Akt信号通路与肝星状细胞活化的关系及其在放射性肝纤维化中的作用. 中华放射医学与防护杂志,2015,35(9):652-655. [28] 管洪庚,徐永锋,兰晶.血小板源生长因子通过PI3K/AKT途径促肝星状细胞增殖与Ⅰ型胶原合成.中华实验外科杂志,2007,24(11):1332-1334. [29] Jatiani SS,Baker SJ,Silverman LR,et al. JAK/STAT pathways in cytokine signaling and myeloproliferative disorder:approaches for targeted therapies. Genes Cancer,2010,1(10):979-993. [30] Lakner AM,Moore CC,Gulledge AA,et al. Daily genetic profiling indicates JAK/STAT signaling promotes early hepatic stellate cell transdifferentiation.World J Gastroenterol,2010,16(40):5047-5056. [31] Cipolletta E,Monaco S,Maione AS,et al. Calmodulin-dependent kinase Ⅱmediates vascular smooth muscle cell proliferation and is potentiated by extracellular signal regulated kinase.Endocrinology,2010,151(6):2747-2759. [32] 陈莲香,舒建昌. PDGF 与肝纤维化关系的研究新进展. 胃肠病学和肝病学杂志,2011,20(1):95-98. [33] Kaji K,Yoshiji H,Ikenaka Y,et al. DipeptidyI peptidase-4 inhibitor attenuates hepatic fibrosis via suppression of activated hepatic stellate cell in rats. J Gastroenterol,2014,49(3):481-491. [34] 陈暑波,冯军,赵帆,等. 大鼠急性放射性肝损伤TGF-β1和α-SMA的表达变化. 中国现代医学杂志,2015,25(1):12-15. [35] Chun J,Zhang X,Jin L,et al.Protective effects of caffeic acid phenethyl ester against acute radiation-induced hepatic injury in rats.Environ Toxicol Pharmacol,2015,39(2):683-689. [36] Du SS,Qiang M,Zeng ZC,et al. Radiation-induced liver fibrosis is mitigated by gene therapy inhibiting transforming growth factor-β signaling in the rat.Int J Radiat Oncol Biol Phys,2010,78(5):1513-1523. |
Viewed | ||||||
Full text |
|
|||||
Abstract |
|
|||||