Journal of Practical Hepatology ›› 2022, Vol. 25 ›› Issue (2): 157-160.doi: 10.3969/j.issn.1672-5069.2022.02.002
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Zhou Ping, Yao Dengfu, Yao Min
Received:
2021-11-12
Online:
2022-03-10
Published:
2022-03-15
Zhou Ping, Yao Dengfu, Yao Min. Hepatocyte malignant transformation in circumstances of nonalcoholic fatty liver diseases[J]. Journal of Practical Hepatology, 2022, 25(2): 157-160.
[1] Huang DQ, El-Serag HB, Loomba R.Global epidemiology of NAFLD-related HCC: trends, predictions, risk factors and prevention. Nat Rev Gastroenterol Hepatol, 2021,18(4):223-238. [2] Zou ZY, Zeng J, Ren TY, et al. Efficacy of intragastric balloons in the markers of metabolic dysfunction-associated fatty liver disease: Results from meta-analyses. J Clin Transl Hepatol, 2021, 9(3):353-363. [3] Bence KK, Birnbaum MJ.Metabolic drivers of non-alcoholic fatty liver disease. Mol Metab, 2021, 50:101143. [4] Prasun P, Ginevic I, Oishi K.Mitochondrial dysfunction in nonalcoholic fatty liver disease and alcohol related liver disease. Transl Gastroenterol Hepatol, 2021,6:4. [5] Choudhary NS, Duseja A.Genetic and epigenetic disease modifiers: non-alcoholic fatty liver disease (NAFLD) and alcoholic liver disease (ALD). Transl Gastroenterol Hepatol, 2021, 6:2. DOI:10.21037/tgh.2019.09.06 [6] Geh D, Manas DM, Reeves HL.Hepatocellular carcinoma in non-alcoholic fatty liver disease-a review of an emerging challenge facing clinicians. Hepatobiliary Surg Nutr, 2021, 10(1): 59-75. [7] Eguchi Y, Wong G, Lee IH, et al.Hepatocellular carcinoma and other complications of non- alcoholic fatty liver disease and non-alcoholic steatohepatitis in Japan: A structured review of published works. Hepatol Res, 2021, 51(1):19-30. [8] Feng G, Li XP, Niu CY, et al. Bioinformatics analysis reveals novel core genes associated with nonalcoholic fatty liver disease and nonalcoholic steatohepatitis. Gene, 2020, 742: 144549. [9] Jiang ZY, Zhou Y, Zhou L,et al. Identification of key genes and immune infiltrate in nonalcoholic steatohepatitis: A bioinformatic analysis. Biomed Res Int, 2021, 2021:7561645. [10] Endo S, Matsunaga T, Nishinaka T.The role of AKR1B10 in physiology and patho- physiology. Metabolites, 2021,11(6):332. [11] Orci LA, Sanduzzi-Zamparelli M, Caballol B, et al.Incidence of hepatocellular carcinoma in patients with nonalcoholic fatty liver disease: A systematic review, meta-analysis, and meta- regression. Clin Gastroenterol Hepatol. 2021, S1542-3565(21)00505-X. [12] Barrow F, Khan S, Wang H,et al. The emerging role of B cells in the pathogenesis of NAFLD. Hepatology, 2021[ahead of print]. [13] Khan O.Giles JR, McDonald S, et al. TOX transcriptionally and epigenetically programs CD8+T cell exhaustion. Nature, 2019,571:211–218. [14] Pfister D, Núñez NG, Inyol R, et al. NASH limits anti-tumour surveillance in immuno-therapy-treated HCC. Nature, 2021,592(7854):450-456. [15] Raza S, Rajak S, Upadhyay A, et al.Current treatment paradigms and emerging therapies for NAFLD/NASH. Front Biosci (Landmark Ed). 2021; 26:206-237. [16] Ma C, Kesarwala AH, Eggert T, et al.NAFLD causes selective CD4 (+) T lymphocyte loss and promotes hepatocarcinogenesis. Nature 2016, 531(7593):253-257. [17] Wang T, Sun G, Wang Y, et al.The immunoregulatory effects of CD8 T-cell-derived perforin on diet-induced nonalcoholic steatohepatitis. FASEB J, 2019, 33(7):8490-8503. [18] Van Herck MA, Weyler J, Kwanten WJ, et al.The differential roles of T cells in non- alcoholic fatty liver disease and obesity. Front Immunol, 2019,10:82. [19] Van Herck MA, Vonghia L, Kwanten WJ, et al.Adoptive cell transfer of regulatory T cells exacerbates hepatic steatosis in high-fat high-fructose diet-fed mice. Front Immunol, 2020, 11:1711. [20] S'widerska M, Jaroszewicz J, Stawicka A, et al. The interplay between Th17 and T-regulatory responses as well as adipokines in the progression of non-alcoholic fatty liver disease. Clin Exp Hepatol, 2017, 3(3):127-134. [21] He B, Wu L, Xie W, et al.The imbalance of Th17/Treg cells is involved in the progression of nonalcoholic fatty liver disease in mice. BMC Immunol, 2017,18(1):33. [22] Durand M, Coué M, Croyal M, et al.Changes in key mitochondrial lipids accompany mito- chondrial dysfunction and oxidative stress in NAFLD. Oxid Med Cell Longev, 2021, 2021: 9986299. [23] Dabravolski SA, Bezsonov EE, Baig MS, et al.Mitochondrial mutations and genetic factors determining NAFLD risk. Int J Mol Sci, 2021, 22(9):4459. [24] Ajaz S, McPhail MJ, Gnudi L, et al.Mitochondrial dysfunction as a mechanistic biomarker in patients with non-alcoholic fatty liver disease (NAFLD). Mitochondrion 2021,57:119-130. [25] Meex RCR, Blaak EE.Mitochondrial dysfunction is a key pathway that links saturated fat intake to the development and progression of NAFLD. Mol Nutr Food Res, 2021, 65(1): e1900942. [26] Xiao WC, Ren M, Can Zhang C, et al. Amelioration of nonalcoholic fatty liver disease by hepatic stimulator substance via preservation of carnitine palmitoyl transferase-1 activity. Am J Physiol Cell Physiol, 2015, 309(4):C215-227. [27] Gu JJ, Yao M, Yang J, et al.Mitochondrial carnitine palmitoyl transferase-II inactivity aggravates lipid accumulation in rat hepatocarcinogenesis. World J Gastroenterol, 2017, 23 (2): 256-264. [28] Xu D, Tian Y, Xia Q, et al.The cGAS-STING pathway: Novel perspectives in liver diseases. Front Immunol, 2021, 12:682736. [29] Li A, Yi M, Qin S, et al. Activating cGAS-STING pathway for the optimal effect of cancer immunotherapy. J Hematol Oncol, 2019,12: 35. [30] Longo M, Paolini E, Meroni M, et al.Remodeling of mitochondrial plasticity: The key switch from NAFLD/NASH to HCC. Int J Mol Sci, 2021, 22(8):4173. [31] Giraud J, Saleh M.Host-microbiota interactions in liver inflammation and cancer. Cancers (Basel), 2021,13(17):4342. [32] Wu L, Li J, Feng J, et al.Crosstalk between PPARs and gut microbiota in NAFLD. Biomed Pharmacother, 2021, 136:111255. |
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